The physiological and ventilatory responses to repeated 60 s sprints following sodium citrate ingestion

G. Cox, D. G. Jenkins

Research output: Contribution to journalArticleResearchpeer-review

14 Citations (Scopus)

Abstract

This study examined the influence of sodium citrate on changes in selected blood, ventilatory and performance variables in response to intermittent sprint exercise. Eight moderately active male students completed three tests over a 6 day experimental period. The first test involved incremental exercise to determine VO2 max, while the second and third tests were identical in nature and involved five 60 s sprints cycling against 0.075 kg kg-1 body mass (BM); each of the five sprints was separated by 5 min passive seated recovery. Three days separated the VO2 max test and first interval test, while a further 3 days elapsed between the first and second interval tests. Ninety minutes prior to each interval test, the subjects consumed either a solution of sodium citrate (0.5 g kg-1 BM.) or a placebo solution (1 g of calcium carbonate and 4 mg of sodium chloride). These were randomly administered in a double-blind crossover procedure so that every subject consumed each solution prior to the interval test over the 6 day period. Measures of work, VE, VO2, VCO2, post-exercise plasma lactate, and changes in both venous blood pH and venous blood bicarbonate (Equation found ) were measured during each interval test. Although analysis of variance failed to identify differences in performance between the two solutions, both exercise VCO2 and changes in venous blood (Equation found) were higher in the citrate condition (P < 0.05). In addition, both peak post-exercise plasma lactate concentrations and post-exercise venous blood pH were significantly higher following citrate ingestion. Although these data are consistent with greater clearance of lactate and H+ from the active muscle cells following citrate ingestion, performance between the two trials was the same. Nausea experienced by all but one subject following citrate ingestion emerged as the most likely factor negating the ergogenic potential of the sodium citrate. The data have shown that citrate ingested prior to five 60 s sprints induced significant changes in selected ventilatory and blood variables but failed to influence work output.

Original languageEnglish
Pages (from-to)469-475
Number of pages7
JournalJournal of Sports Sciences
Volume12
Issue number5
DOIs
Publication statusPublished - 1 Jan 1994
Externally publishedYes

Fingerprint

Citric Acid
Eating
Exercise
Lactic Acid
Calcium Carbonate
Bicarbonates
Sodium Chloride
Muscle Cells
Nausea
sodium citrate
Analysis of Variance
Placebos
Students

Cite this

@article{92b816c62a0347f8904b434167cb754d,
title = "The physiological and ventilatory responses to repeated 60 s sprints following sodium citrate ingestion",
abstract = "This study examined the influence of sodium citrate on changes in selected blood, ventilatory and performance variables in response to intermittent sprint exercise. Eight moderately active male students completed three tests over a 6 day experimental period. The first test involved incremental exercise to determine VO2 max, while the second and third tests were identical in nature and involved five 60 s sprints cycling against 0.075 kg kg-1 body mass (BM); each of the five sprints was separated by 5 min passive seated recovery. Three days separated the VO2 max test and first interval test, while a further 3 days elapsed between the first and second interval tests. Ninety minutes prior to each interval test, the subjects consumed either a solution of sodium citrate (0.5 g kg-1 BM.) or a placebo solution (1 g of calcium carbonate and 4 mg of sodium chloride). These were randomly administered in a double-blind crossover procedure so that every subject consumed each solution prior to the interval test over the 6 day period. Measures of work, VE, VO2, VCO2, post-exercise plasma lactate, and changes in both venous blood pH and venous blood bicarbonate (Equation found ) were measured during each interval test. Although analysis of variance failed to identify differences in performance between the two solutions, both exercise VCO2 and changes in venous blood (Equation found) were higher in the citrate condition (P < 0.05). In addition, both peak post-exercise plasma lactate concentrations and post-exercise venous blood pH were significantly higher following citrate ingestion. Although these data are consistent with greater clearance of lactate and H+ from the active muscle cells following citrate ingestion, performance between the two trials was the same. Nausea experienced by all but one subject following citrate ingestion emerged as the most likely factor negating the ergogenic potential of the sodium citrate. The data have shown that citrate ingested prior to five 60 s sprints induced significant changes in selected ventilatory and blood variables but failed to influence work output.",
author = "G. Cox and Jenkins, {D. G.}",
year = "1994",
month = "1",
day = "1",
doi = "10.1080/02640419408732197",
language = "English",
volume = "12",
pages = "469--475",
journal = "Journal of Sports Sciences",
issn = "0264-0414",
publisher = "Taylor & Francis",
number = "5",

}

The physiological and ventilatory responses to repeated 60 s sprints following sodium citrate ingestion. / Cox, G.; Jenkins, D. G.

In: Journal of Sports Sciences, Vol. 12, No. 5, 01.01.1994, p. 469-475.

Research output: Contribution to journalArticleResearchpeer-review

TY - JOUR

T1 - The physiological and ventilatory responses to repeated 60 s sprints following sodium citrate ingestion

AU - Cox, G.

AU - Jenkins, D. G.

PY - 1994/1/1

Y1 - 1994/1/1

N2 - This study examined the influence of sodium citrate on changes in selected blood, ventilatory and performance variables in response to intermittent sprint exercise. Eight moderately active male students completed three tests over a 6 day experimental period. The first test involved incremental exercise to determine VO2 max, while the second and third tests were identical in nature and involved five 60 s sprints cycling against 0.075 kg kg-1 body mass (BM); each of the five sprints was separated by 5 min passive seated recovery. Three days separated the VO2 max test and first interval test, while a further 3 days elapsed between the first and second interval tests. Ninety minutes prior to each interval test, the subjects consumed either a solution of sodium citrate (0.5 g kg-1 BM.) or a placebo solution (1 g of calcium carbonate and 4 mg of sodium chloride). These were randomly administered in a double-blind crossover procedure so that every subject consumed each solution prior to the interval test over the 6 day period. Measures of work, VE, VO2, VCO2, post-exercise plasma lactate, and changes in both venous blood pH and venous blood bicarbonate (Equation found ) were measured during each interval test. Although analysis of variance failed to identify differences in performance between the two solutions, both exercise VCO2 and changes in venous blood (Equation found) were higher in the citrate condition (P < 0.05). In addition, both peak post-exercise plasma lactate concentrations and post-exercise venous blood pH were significantly higher following citrate ingestion. Although these data are consistent with greater clearance of lactate and H+ from the active muscle cells following citrate ingestion, performance between the two trials was the same. Nausea experienced by all but one subject following citrate ingestion emerged as the most likely factor negating the ergogenic potential of the sodium citrate. The data have shown that citrate ingested prior to five 60 s sprints induced significant changes in selected ventilatory and blood variables but failed to influence work output.

AB - This study examined the influence of sodium citrate on changes in selected blood, ventilatory and performance variables in response to intermittent sprint exercise. Eight moderately active male students completed three tests over a 6 day experimental period. The first test involved incremental exercise to determine VO2 max, while the second and third tests were identical in nature and involved five 60 s sprints cycling against 0.075 kg kg-1 body mass (BM); each of the five sprints was separated by 5 min passive seated recovery. Three days separated the VO2 max test and first interval test, while a further 3 days elapsed between the first and second interval tests. Ninety minutes prior to each interval test, the subjects consumed either a solution of sodium citrate (0.5 g kg-1 BM.) or a placebo solution (1 g of calcium carbonate and 4 mg of sodium chloride). These were randomly administered in a double-blind crossover procedure so that every subject consumed each solution prior to the interval test over the 6 day period. Measures of work, VE, VO2, VCO2, post-exercise plasma lactate, and changes in both venous blood pH and venous blood bicarbonate (Equation found ) were measured during each interval test. Although analysis of variance failed to identify differences in performance between the two solutions, both exercise VCO2 and changes in venous blood (Equation found) were higher in the citrate condition (P < 0.05). In addition, both peak post-exercise plasma lactate concentrations and post-exercise venous blood pH were significantly higher following citrate ingestion. Although these data are consistent with greater clearance of lactate and H+ from the active muscle cells following citrate ingestion, performance between the two trials was the same. Nausea experienced by all but one subject following citrate ingestion emerged as the most likely factor negating the ergogenic potential of the sodium citrate. The data have shown that citrate ingested prior to five 60 s sprints induced significant changes in selected ventilatory and blood variables but failed to influence work output.

UR - http://www.scopus.com/inward/record.url?scp=0028043138&partnerID=8YFLogxK

U2 - 10.1080/02640419408732197

DO - 10.1080/02640419408732197

M3 - Article

VL - 12

SP - 469

EP - 475

JO - Journal of Sports Sciences

JF - Journal of Sports Sciences

SN - 0264-0414

IS - 5

ER -