Computational cognitive models of prefrontal-striatal-hippocampal interactions in Parkinson's disease and schizophrenia

Keyphrases

• Parkinson's Disease 100%
• Schizophrenia 100%
• Striatal 100%
• Computational Cognitive Model 100%
• Prefrontal Cortex 80%
• Basal Ganglia 60%
• Levodopa 40%
• Antipsychotics 40%
• Dopamine Agonist 40%
• Hippocampal Circuit 40%
• Cognitive Deficits 20%
• Clinical Data 20%
• Neurological Disorders 20%
• Functional Role 20%
• Dissociation 20%
• Basal Ganglia Circuits 20%
• Physiological Data 20%
• Extended Model 20%
• Hippocampus 20%
• Negative Symptoms 20%
• Dopamine Level 20%
• Psychiatric Disorders 20%
• Cognitive Dysfunction 20%
• Brain Disorders 20%
• D2 Dopamine Receptor 20%
• Hippocampal Dysfunction 20%
• Stimulus-response Learning 20%
• Frontostriatal Circuits 20%
• Model Limitations 20%
• D1 Dopamine Receptor 20%
• Multidimensional Stimuli 20%
• Uniform Effect 20%
• Stimulus Selection 20%
• Representation Learning 20%
• Modelling Challenges 20%

Neuroscience

• Parkinson's Disease 100%
• Basal Ganglia 80%
• Prefrontal Cortex 80%
• Cognitive Disorder 40%
• L-DOPA 40%
• Dopamine Agonist 40%
• Nervous System Disorder 20%
• Stimulus Response 20%
• Mental Disorder 20%
• Dopamine Receptor D2 20%
• Hippocampus 20%
• Negative Syndrome 20%
• Brain Disease 20%
• Dopamine Receptor D1 20%