Carbachol-induced increases in spontaneous contractile activity are mediated through the M3- muscarinic receptor

Research output: Contribution to conferenceAbstractResearch

Abstract

It is understood that the urothelium (urothelium-suburothelium) influences the detrusor muscle, yet the extent is
uncertain. There is a possibility that abnormal activity of the urothelium can induce bladder dysfunctions such as
detrusor overactivity. Upon activation with carbachol, the frequency of urothelial spontaneous contractions is
increased. The aim of this study was to classify which receptor is activated by carbachol.
Original languageEnglish
Publication statusPublished - 2010
Event2nd National Symposium on Advances in Urogenital and Gut Research - Robina, Australia
Duration: 3 Aug 2010 → …

Conference

Conference2nd National Symposium on Advances in Urogenital and Gut Research
CountryAustralia
CityRobina
Period3/08/10 → …

Fingerprint

Muscarinic M3 Receptors
Urothelium
Carbachol
Urinary Bladder
Muscles

Cite this

Moro, C., & Chess-Williams, R. (2010). Carbachol-induced increases in spontaneous contractile activity are mediated through the M3- muscarinic receptor. Abstract from 2nd National Symposium on Advances in Urogenital and Gut Research , Robina, Australia.
Moro, Christian ; Chess-Williams, Russ. / Carbachol-induced increases in spontaneous contractile activity are mediated through the M3- muscarinic receptor. Abstract from 2nd National Symposium on Advances in Urogenital and Gut Research , Robina, Australia.
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title = "Carbachol-induced increases in spontaneous contractile activity are mediated through the M3- muscarinic receptor",
abstract = "It is understood that the urothelium (urothelium-suburothelium) influences the detrusor muscle, yet the extent isuncertain. There is a possibility that abnormal activity of the urothelium can induce bladder dysfunctions such asdetrusor overactivity. Upon activation with carbachol, the frequency of urothelial spontaneous contractions isincreased. The aim of this study was to classify which receptor is activated by carbachol.",
author = "Christian Moro and Russ Chess-Williams",
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language = "English",
note = "2nd National Symposium on Advances in Urogenital and Gut Research ; Conference date: 03-08-2010",

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Moro, C & Chess-Williams, R 2010, 'Carbachol-induced increases in spontaneous contractile activity are mediated through the M3- muscarinic receptor' 2nd National Symposium on Advances in Urogenital and Gut Research , Robina, Australia, 3/08/10, .

Carbachol-induced increases in spontaneous contractile activity are mediated through the M3- muscarinic receptor. / Moro, Christian; Chess-Williams, Russ.

2010. Abstract from 2nd National Symposium on Advances in Urogenital and Gut Research , Robina, Australia.

Research output: Contribution to conferenceAbstractResearch

TY - CONF

T1 - Carbachol-induced increases in spontaneous contractile activity are mediated through the M3- muscarinic receptor

AU - Moro, Christian

AU - Chess-Williams, Russ

PY - 2010

Y1 - 2010

N2 - It is understood that the urothelium (urothelium-suburothelium) influences the detrusor muscle, yet the extent isuncertain. There is a possibility that abnormal activity of the urothelium can induce bladder dysfunctions such asdetrusor overactivity. Upon activation with carbachol, the frequency of urothelial spontaneous contractions isincreased. The aim of this study was to classify which receptor is activated by carbachol.

AB - It is understood that the urothelium (urothelium-suburothelium) influences the detrusor muscle, yet the extent isuncertain. There is a possibility that abnormal activity of the urothelium can induce bladder dysfunctions such asdetrusor overactivity. Upon activation with carbachol, the frequency of urothelial spontaneous contractions isincreased. The aim of this study was to classify which receptor is activated by carbachol.

M3 - Abstract

ER -

Moro C, Chess-Williams R. Carbachol-induced increases in spontaneous contractile activity are mediated through the M3- muscarinic receptor. 2010. Abstract from 2nd National Symposium on Advances in Urogenital and Gut Research , Robina, Australia.